Fig. 1From: Acute upregulation of neuronal mitochondrial type-1 cannabinoid receptor and it’s role in metabolic defects and neuronal apoptosis after TBITBI induced acute upregulation of mtCB1 and plasma membrane CB1 24 h post injury. Western blot analysis of mtCB1 and plasma membrane CB1 24 h post injury showed CB1 increased in mitochondria separated from cultured neurons and wild type mice after injury but not CB1−/− mice (a, b). CB1 relatively slightly increased in plasma membrane fractions separated from cultured neurons and wild type mice after injury but not CB1−/− mice (c, d). Cadherin is a plasma membrane protein, Tom20 is a mitochondrial protein. Values were expressed as mean ± SE (*p < 0.05 versus sham, ***p < 0.001 versus sham)Back to article page