A joint publication of BMC, part of Springer Nature, and the Editorial Group of Molecular Brain since 2008.
Molecular Brain is affiliated with the Association for the Study of Neurons and Disease (AND).
Page 20 of 20
We have previously demonstrated that PDGF receptor activation indirectly inhibits N-methyl-D-aspartate (NMDA) currents by modifying the cytoskeleton. PDGF receptor ligand is also neuroprotective in hippocampal...
Citation: Molecular Brain 2008 1:20
Temporal control of brain and behavioral states emerges as a consequence of the interaction between circadian and homeostatic neural circuits. This interaction permits the daily rhythm of sleep and wake, regul...
Citation: Molecular Brain 2008 1:19
AChR is concentrated at the postjunctional membrane at the neuromuscular junction. However, the underlying mechanism is unclear. We show that α-actinin, a protein known to cross-link F-actin, interacts with ra...
Citation: Molecular Brain 2008 1:18
α-synuclein (α-syn) is a main component of Lewy bodies (LB) that occur in many neurodegenerative diseases, including Parkinson's disease (PD), dementia with LB (DLB) and multi-system atrophy. α-syn mutations o...
Citation: Molecular Brain 2008 1:17
Phenylethanolamines selectively bind to NR2B subunit-containing N-methyl-D-aspartate-subtype of ionotropic glutamate receptors and negatively modulate receptor activity. To investigate the structural and function...
Citation: Molecular Brain 2008 1:16
Deposition of amyloid-β protein (Aβ) is a major pathological hallmark of Alzheimer's disease (AD). Aβ is generated from γ-secretase cleavage of amyloid precursor protein (APP). In addition to APP, γ-secretase ...
Citation: Molecular Brain 2008 1:15
Ca2+-activated Cl- channels (CaCCs) participate in many important physiological processes. However, the lack of effective and selective blockers has hindered the study of these channels, mostly due to the lack of...
Citation: Molecular Brain 2008 1:14
Although long-term potentiation (LTP) of synaptic strength is very persistent, current studies have provided evidence that various manipulations or pharmacological treatment when applied shortly after LTP indu...
Citation: Molecular Brain 2008 1:13
In previous work, we investigated dieldrin cytotoxicity and signaling cell death mechanisms in dopaminergic PC12 cells. Dieldrin has been reported to be one of the environmental factors correlated with Parkins...
Citation: Molecular Brain 2008 1:12
Schizophrenia is a complex genetic disorder caused by multiple genetic and environmental factors. The dystrobrevin-binding protein 1 (DTNBP1: dysbindin-1) gene is a major susceptibility gene for schizophrenia....
Citation: Molecular Brain 2008 1:11
Several myelin-associated factors that inhibit axon growth of mature neurons, including Nogo66, myelin-associated glycoprotein (MAG) and oligodendrocyte myelin glycoprotein (OMgp), can associate with a common ...
Citation: Molecular Brain 2008 1:10
Long-term potentiation (LTP) in the hippocampal CA1 region requires the activation of N-methyl-D-aspartate receptors (NMDARs). Studies using genetic and pharmacological approaches have reported inconsistent re...
Citation: Molecular Brain 2008 1:9
The midbrain dopaminergic (mDA) neurons of the substantia nigra and the ventral tegmental area play a fundamental role in the control of voluntary movement and the regulation of emotion, and are severely affec...
Citation: Molecular Brain 2008 1:8
Calpastatin is an endogenous inhibitor of calpain, intracellular calcium-activated protease. It has been suggested to be involved in molecular mechanisms of long-term plasticity and excitotoxic pathways. Howev...
Citation: Molecular Brain 2008 1:7
Elucidating the neural and genetic factors underlying psychiatric illness is hampered by current methods of clinical diagnosis. The identification and investigation of clinical endophenotypes may be one soluti...
Citation: Molecular Brain 2008 1:6
Using an endophenotype-driven screen, a new study finds that α-calcium/calmodulin kinase II mutant mice exhibit a range of behavioral abnormalities related to schizophrenia. Perhaps most strikingly, this clust...
Citation: Molecular Brain 2008 1:5
Diurnal rhythm-mediated endogenous cortisol levels in humans are characterised by a peak in secretion after awakening that declines throughout the day to an evening trough. However, a significant proportion of...
Citation: Molecular Brain 2008 1:4
Whereas the induction of short-term memory involves only covalent modifications of constitutively expressed preexisting proteins, the formation of long-term memory requires gene expression, new RNA, and new pr...
Citation: Molecular Brain 2008 1:3
Morphine is widely used to treat chronic pain, however its utility is hindered by the development of tolerance to its analgesic effects. While N-methyl-D-aspartate (NMDA) receptors are known to play roles in m...
Citation: Molecular Brain 2008 1:2
We are delighted to announce the arrival of a brand new journal dedicated to the ever-expanding field of neuroscience. Molecular Brain is a peer-reviewed, open-access online journal that aims at publishing high q...
Citation: Molecular Brain 2008 1:1
A joint publication of BMC, part of Springer Nature, and the Editorial Group of Molecular Brain since 2008.
Molecular Brain is affiliated with the Association for the Study of Neurons and Disease (AND).
Speed
31 days to first decision for reviewed manuscripts only
26 days to first decision for all manuscripts
78 days from submission to acceptance
17 days from acceptance to publication
Citation Impact
4.486 - 2-year Impact Factor
4.226 - 5-year Impact Factor
1.087 - Source Normalized Impact per Paper (SNIP)
1.945 - SCImago Journal Rank (SJR)
Usage
379,102 downloads
706 Altmetric mentions
Molecular Brain has partnered with Publons to give you official recognition for your contribution to peer review. Create a free profile to have your reviews automatically added.