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Figure 1 | Molecular Brain

Figure 1

From: Activation of c-jun N-terminal kinase in spinal cord contributes to breast cancer induced bone pain in rats

Figure 1

Time course of pJNK upregulation on the ipsilateral side of L4-L5 spinal cord after intra-tibial inoculation with carcinoma cells. (A-B) Representative Western blots of pJNK1/2 and GAPDH from one membrane. (C-D) Density of pJNK1/2 levels on the ipsilateral side of L4-L5 spinal cord. pJNK1/2 levels were normalized against GAPDH levels and expressed as fold increase, compared with naïve. (* p < 0.05 and ** p < 0.01, one-way ANOVA, Mean ± SEM n = 4)

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